Evolution of phenotypic traits

Quantitative genetics

Very few phenotypic traits are controlled by one locus, as in our previous discussion of genetics and evolution

Quantitative genetics considers characters that are controlled by more than one locus

Multilocus systems

Definitions

In “multilocus systems,” a phenotypic trait is influenced by more than one locus

Each locus might have alternative alleles

Diverse alleles and loci produce phenotypic “polymorphism”

Different loci can be on one chromosome (“linked”) or scattered

Quantitative genetics

The study of traits affected by many genes and the environment

Polygenic traits show continuous variation

Most phenotypes lack clear categories

Living histogram

Living histogram

Swallowtail polymorphism

Heliconius mimicry

Multiple loci

H. erato (15) and H. melpomene (12)

Loci are scattered on different chromosomes – not linked

Mating between forms produces diverse (and unsuccessful) intermediates

What are quantitative traits (QT)?

Continuous variation (lack of discrete categories)

Influenced by:

genotype at many different loci

environmental (developmental) influences

gene-environment interactions

What does this experiment tell us?

Evolution occurs in quantitative traits..

Examples

Behavioral

Physiological

Morphological

Evolution in behavior

Critical photoperiod for entering diapause has changed in introduced populations

Evolution in physiology

Metal tolerant plants

Cost of tolerance detected

Evolution in morphology

Soapberry bugs

How do we add this complexity to models of population genetics?

It depends

How many loci are involved?

Do the loci assort independently, or as a group?

How are genes influencing quantitative traits identified?

QTL mapping

More complex than “on-off” or “single-gene” approach

Measures association between variation in a phenotype and a genetic marker

Reveals

Number of loci influencing a quantitative trait

Magnitude of their effects on the quantitative trait

Location in the genome

Juenger et al., 2005. Quantitative trait loci mapping of floral and leaf morphology traits in Arabidopsis thaliana . . .. Evolution & Development 7:259-271.

Genomic positions of QTL for floral and leaf characters – Juenger et al. 2005

General conclusions about QTL

Variation may be influenced by dozens of loci

53 QTL for Drosophila bristle number

Loci vary in intensity of effect

Diverse genetic mechanisms for affecting variation

Additive effects

Epistatic effects

Gene-environment interactions

Linkage disequilibrium

What is linkage disequilibrium?

Why is this phenomenon important?

How is linkage disequilibrium measured?

What factors influence change in linkage disequilibrium?

How are linkage disequilibrium and natural selection related?

What is linkage disequilibrium?

Addresses relations among different genes (loci)

Genomes are organized by chromosome structure

Law of Independent Assortment

of alleles from different loci

does not apply to different loci physically linked by proximity on the same chromosome

Linkage disequilibrium defined

What is linkage equilibrium?

When genotypes of one locus are distributed independently of genotypes at another locus, the two loci are in linkage equilibrium

A “null model” for whether gametes tend to have similar combinations of alleles (compared to random combinations)

What is linkage disequilibrium

anything else

Haplotype: the arrangement of alleles along a chromosome

How is linkage disequilibrium measured?

Count each haplotype in the population

e.g., AB Ab aB ab

Calculate frequencies (the proportion of all chromosomes represented by each haplotype)

gAB gAb gaBgab

Coefficient of Linkage Disequilibrium = D

D = gAB*gab – gAb* gaB

Linkage disequilibrium tends to decline

What reduces physical linkage?

Crossing over during meiosis

Sexual reproduction is required

Genetic recombination results from

shuffling of chromosomes

crossing over within chromosomes

Recombination rates affect LD breakdown

An experimental test of L.-D. theory

Why is L.-D. observed in nature?

Nonrandom mating

New mutations arising

Recent population formation

Low recombination

Genetic drift

Selection on multilocus genotypes

How does L.-D. influence selection?

What if . . . a new, beneficial mutation is linked with a deleterious allele?

What if . . . a new, nasty mutation is linked with a beneficial allele?

Can single-locus models predict evolutionary change?

Measuring variation

A review and revision

Measure of variation

Variance is measure of variation in natural populations

Average of squared deviations from the mean of a sample

Standard deviation is the square root of the variance

If a variable has a normal distribution, 68% of the observations will be within 1 SD, 96% in 2 SD and 99.7 in 3 SD

Variation in phenotype is due to genes AND environment:

For one locus,

Midpoint between two homozygotes as a point of reference

Mean phenotype of A1A1 individuals is A+a, and mean phenotype of A2A2 individuals is A–a.

a is the additive effect of an allele

If the inheritance of the phenotype is completely additive, then the heterozygote is exactly in between that of the two homozygotes

Phenotypic variance within populations is the environmental variance, VE

Additive genetic variance

Component of genetic variance denoted VA

Genetic variance includes other variance as well, but we will not discuss here

VA depends on magnitude of a and genotype frequencies

Variance is lower if one genotype is most common

At one locus, VA = 2pqa2

At several loci,

Average phenotype of any particular genotype is the sum of the phenotypic values of each loci

VA is the sum of the additive genetic variance contributed by each of the loci

Additive genetic variance

The additive effects of alleles are responsible for the degree of similarity between parents and offspring

Therefore, are the basis for response to selection within populations

Expected average phenotype of a brood of offspring equals the average of their parents’ phenotypes

Additive genetic inheritance allows a response to selection

Heritability in the narrow sense

Heritability is determined by the additive genetic variance (depends on allele frequencies) and environmental variance (depends on environment)

hN2 = VA /(VG + VE)

Compare to heritability in the broad sense we learned earlier

h2 = VG /(VG + VE)

hN2

hN2 is the slope of the regression of offspring phenotypes on the average of the parents of each brood of offspring

Heritability

Narrow sense heritability = h2 = VA/VP

= VA

(VA + VD + VI + VE)

VP = variation in phenotype

VA = additive effects of genes

VD = dominance effects

VI = interaction between genes (epistasis)

VE = environment effects

Heritability

Broad sense heritability = H = VG/VP

Variation due to dominance and interactions not truly heritable

H is seldom used, ‘heritability’ is usually heritability in the narrow sense (h2)